DefinitionThis section has been translated automatically.
EtiopathogenesisThis section has been translated automatically.
Triggered by drugs, e.g. systemic glucocorticoids, nicotinic acid, estrogens, insulin, methyltestosterone, oral contraceptives, fusidic acid, folic acid antagonists. Interactions between these drugs with lipid metabolism are postulated. Furthermore, an overstimulation of receptor tyrosine kinases by the above mentioned drugs might play a role.
TherapyThis section has been translated automatically.
Outgoing links (1)Acanthosis nigricans (overview);
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